The Luminescent Oligothiophene p-FTAA Converts Toxic Aβ1–42 Species into Nontoxic Amyloid Fibers with Altered Properties.
Journal of Biological Chemistry; doi: 10.1074/jbc.M115.696229..
PHARMACEUTICAL COMPOSITION FOR TREATING OR PREVENTING DEGENERATIVE BRAIN DISEASE COMPRISING MULTI-TARGETING COMPOUNDS
freepatentsonline; .
Aβ-Induced Insulin Resistance and the Effects of Insulin on the Cholesterol Synthesis Pathway and Aβ Secretion in Neural Cells
Springer Link; DOI: 10.1007/s12264-016-0034-9.
Ketones block amyloid entry and improve cognition in an Alzheimer's model.
Neurobiol Aging.; doi: 10.1016/j.neurobiolaging.2015.11.018..
ABCA7 Mediates Phagocytic Clearance of Amyloid-β in the Brain.
Journal of Alzheimer’s Disease; doi: 10.3233/JAD-160456..
The involvement of dityrosine crosslinking in α-synuclein assembly and deposition in Lewy Bodies in Parkinson’s disease
Nature.com Scientific Reports; 10.1038/srep39171.
Regulatory factor X1 depresses ApoE-dependent Aβ uptake by miRNA-124 in microglial response to oxidative stress
Science Direct Neuroscience; dx.doi.org/10.1016/j.neuroscience.2016.12.017.
Multifunctional quinoline-triazole derivatives as potential modulators of amyloid-β peptide aggregation
Journal of Inorganic Biochemistry; https://doi.org/10.1016/j.jinorgbio.2016.04.022.
METHODS AND COMPOSITIONS FOR TREATING ALZHEIMER'S DISEASE.
Free Patents Online; http://www.freepatentsonline.com/y2015/0247857.html.
The Transient Receptor Potential Melastatin 2 (TRPM2) Channel Contributes to β-Amyloid Oligomer-Related Neurotoxicity and Memory Impairment.
The Journal of Neuroscience; doi: 10.1523/JNEUROSCI.4081-14.2015.