Time dynamics of photothermal vs optoacoustic response in mid IR nanoscale biospectroscopy.
Cornell University Library; http://arxiv.org/abs/1509.00726v1.
Physical Chemistry Chemical Physics.
Publishing Journals, books and databases; DOI: 10.1039/C5CP03343H.
Prostaglandin signaling suppresses beneficial microglial function in Alzheimer’s disease models.
The Journal of Clinical Investigation; doi:10.1172/JCI77487.
In Silico and in Vitro Study of Binding Affinity of Tripeptides to Amyloid β Fibrils: Implications for Alzheimer’s Disease.
The Journal of Physical Chemistry; DOI: 10.1021/acs.jpcb.5b00006.
Endogenous Docosahexaenoic Acid (DHA) Prevents Aβ1–42 Oligomer-Induced Neuronal Injury.
Springer US; 10.1007/s12035-015-9224-0.
A Food and Drug Administration-approved Asthma Therapeutic Agent Impacts Amyloid β in the Brain in a Transgenic Model of Alzheimer Disease*.
The Journal of Biological Chemistry; doi: 10.1074/jbc.M114.586602.
Lysosomal Enzyme Glucocerebrosidase Protects against A[Beta]1-42 Oligomer-Induced Neurotoxicity: e0143854.
Plos One; doi:10.1371/journal.pone.0143854.
High-resolution NMR characterization of low abundance oligomers of amyloid-β without purification.
Nature; doi:10.1038/srep11811.
Decrease in Plasma Levels of α-Synuclein Is Evident in Patients with Parkinson’s Disease after Elimination of Heterophilic Antibody Interference.
PLOS ONE; DOI: 10.1371/journal.pone.0123162.
Tissue transglutaminase-catalysed cross-linking induces Apolipoprotein E multimers inhibiting Apolipoprotein E's protective effects towards amyloid-beta-induced toxicity.
JNC Journal of Neurochemistry; DOI: 10.1111/jnc.13203.