Amyloid fibril proteomics of AD brains reveals modifiers of aggregation and toxicity
Molecular Neurodegeneration (2023) 18 (1), 61 DOI: 10.1186/s13024-023-00654-z
Development of an α-synuclein positron emission tomography tracer for imaging synucleinopathies
Cell (2023) 186 (16), 3350-3367.e19 DOI: 10.1016/j.cell.2023.06.004
Blocking microglial activation of reactive astrocytes is neuroprotective in models of Alzheimer’s disease
Scientific Reports; https://doi.org/10.1186/s40478-021-01180-z
Engagement of TREM2 by a novel monoclonal antibody induces activation of microglia and improves cognitive function in Alzheimer’s disease models
Journal of Neuroinflammation;doi: 10.1186/s12974-020-01980-5
Imipramine and olanzapine block apoE4-catalyzed polymerization of Aβ and show evidence of improving Alzheimer’s disease cognition
bioRxiv; https://doi.org/10.1101/2021.03.28.437389
Heterotypic Aβ interactions facilitate amyloid assembly and modify amyloid structure
bioRxiv; https://doi.org/10.1101/2021.04.28.441786
Serotonin Promotes Serum Albumin Interaction with the Monomeric Amyloid β Peptide
International Journal of Molecular Sciences; https://doi.org/10.3390/ijms22115896
APOE2, E3 and E4 differentially modulate cellular homeostasis, cholesterol metabolism and inflammatory response in isogenic iPSC-derived astrocytes
BioRxiv; doi: 10.1016/j.stemcr.2021.11.007
The amyloid-inhibiting NCAM-PrP peptide targets Aβ peptide aggregation in membrane-mimetic environments
iScience; DOI:https://doi.org/10.1016/j.isci.2021.102852
Transcriptome analyses reveal tau isoform-driven changes in transposable element and gene expression
bioRxiv; doi.org/10.1101/2021.04.30.442101