Beta-Amyloid Peptides Publications
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(2021)
Alsalahat, I., Majdoub, Z., Taha, M., et al.
Inhibition of aggregation of amyloid-β through covalent modification with benzylpenicillin; potential relevance to Alzheimer's disease
ScienceDirect; https://doi.org/10.1016/j.bbrep.2021.100943
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(2021)
Sompol, P., Gollihue, J., Kraner, S., et al.
Q134R: Small chemical compound with NFAT inhibitory properties improves behavioral performance and synapse function in mouse models of amyloid pathology
Aging Cell; doi.org/10.1111/acel.13416
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(2021)
Yang, J., Ding, W., Zhu, B., et al.
Reporting amyloid beta levels via bioluminescence imaging with amyloid reservoirs in Alzheimer’s disease models
bioRxiv; doi.org/10.1101/2021.06.05.447217
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(2021)
Vadukul, D., Vrancx, C., Burguet, P., et al.
An evaluation of the self-assembly enhancing properties of cell-derived hexameric amyloid-β
Scientific Reports; doi.org/10.1038/s41598-021-90680-y
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(2021)
Walgrave, H., Balusu, S., Snoeck, S., et al.
Restoring miR-132 expression rescues adult hippocampal neurogenesis and memory deficits in Alzheimer’s disease
Cell Stem Cell; https://doi.org/10.1016/j.stem.2021.05.001
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(2021)
West, T., Kirmess, K., Meyer, M., et al.
A blood-based diagnostic test incorporating plasma Aβ42/40 ratio, ApoE proteotype, and age accurately identifies brain amyloid status: findings from a multi cohort validity analysis
Molecular Neurodegenration; doi: 10.1186/s13024-021-00451-6
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(2021)
Colombo, A., Sadler, R., Llovera., G., et al.
Microbiota-derived short chain fatty acids modulate microglia and promote Aβ plaque deposition
eLife; doi: 10.7554/eLife.59826
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(2021)
Ojo, J., Eisenbaum M., Shackleton, B., et al.
Mural cell dysfunction leads to altered cerebrovascular tau uptake following repetitive head trauma
ScienceDirect; https://doi.org/10.1016/j.nbd.2020.105237
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(2021)
Madhuranthakam, C., Shakeri, A., Rao, P.
Modeling the Inhibition Kinetics of Curcumin, Orange G, and Resveratrol with Amyloid-β Peptide
ACS Omega; doi.org/10.1021/acsomega.1c00610
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(2021)
Shen, K., Reichelt, M., Kyauk, R., et al.
Multiple sclerosis risk gene Mertk is required for microglial activation and subsequent remyelination
Cell Reports; https://doi.org/10.1016/j.celrep.2021.108835
