First amyloid β1‐42 certified reference material for re‐calibrating commercial immunoassays.
Alzheimer’s Association; doi.org/10.1002/alz.12145.
Engulfment of Toxic Amyloid β-protein in Neurons and Astrocytes Mediated by MEGF10.
Neuroscience; doi.org/10.1016/j.neuroscience.2020.07.016.
Interaction between tissue transglutaminase and amyloid-beta: Protein-protein binding versus enzymatic crosslinking.
Analytical Biochemistry; doi.org/10.1016/j.ab.2020.113578.
Metal ion coordination delays amyloid-β peptide self-assembly by forming an aggregation-inert complex.
Journal of Biological Chemistry; doi: 10.1074/jbc.RA120.012738.
Clearance of neurotoxic peptides and proteins by meningothelial cells.
Experimental Cell Research; doi.org/10.1016/j.yexcr.2020.112322.
Reverse engineering synthetic antiviral amyloids.
Nature Communications; doi.org/10.1038/s41467-020-16721-8.
A human induced pluripotent stem cell‐derived cortical neuron human‐on‐a chip system to study Aβ42 and tau‐induced pathophysiological effects on long‐term potentiation.
Alzheimer’s & Dementia; doi: 10.1002/trc2.12029.
Iron chelators inhibit amyloid-β-induced production of lipocalin 2 in cultured astrocytes ..
Neurochemistry International; doi.org/10.1016/j.neuint.2019.104607.
Protecting P-glycoprotein at the Blood-Brain Barrier from Degradation in an Alzheimer’s Disease Mouse Model.
Research Square; DOI: doi.org/10.21203/rs.3.rs-116503/v1.
Increased levels of Stress-inducible phosphoprotein-1 accelerates amyloid-β deposition in a mouse model of Alzheimer’s disease.
Acta Neuropathologica Communications; doi.org/10.1186/s40478-020-01013-5.