Modulation of Neuronal Nicotinic Receptor by Quinolizidine Alkaloids Causes Neuroprotection on a Cellular Alzheimer Model.
J Alzheimers Dis.; doi: 10.3233/JAD-132045.
Modulation of Amyloid-β Protein Precursor Expression by HspB1
Metapress; DOI 10.3233/JAD-140348.
Intracellular selection of peptide inhibitors that target disulphide-bridged Aβ42 oligomers
Wiley Online Library; 10.1002/pro.2509.
The phosphorylation of Hsp20 enhances its association with amyloid-β to increase protection against neuronal cell death
Science Direct; http://dx.doi.org/10.1016/j.mcn.2014.05.002.
Microglial derived tumor necrosis factor-α drives Alzheimer's disease-related neuronal cell cycle events
Neurobiol Dis.; doi: 10.1016/j.nbd.2013.10.007.
Fibrillar β‐amyloid 1‐42 alters cytokine secretion, cholinergic signalling and neuronal differentiation
Wiley Online Library; https://doi.org/10.1111/jcmm.12343.
Genetic inhibition of phosphorylation of the translation initiation factor eIF2α does not block Aβ-dependent elevation of BACE1 and APP levels or reduce amyloid pathology in a mouse model of Alzheimer's disease.
PLoS One; doi: 10.1371/journal.pone.0101643v.
Novel 5-aryloxypyrimidine SEN1576 as a candidate for the treatment of Alzheimer's disease
Cambridge University Press; DOI: http://dx.doi.org/10.1017/S1461145713000886.
Apolipoproteins E and J interfere with amyloid-beta uptake by primary human astrocytes and microglia in vitro
Wiley Online Library; DOI: 10.1002/glia.22619.
Reciprocal modulation of Aβ42 aggregation by copper and homocysteine.
Front Aging Neurosci.; https://doi.org/10.3389/fnagi.2014.00237.