METHODS AND COMPOSITIONS FOR TREATING ALZHEIMER'S DISEASE.
Free Patents Online; http://www.freepatentsonline.com/y2015/0247857.html.
The Transient Receptor Potential Melastatin 2 (TRPM2) Channel Contributes to β-Amyloid Oligomer-Related Neurotoxicity and Memory Impairment.
The Journal of Neuroscience; doi: 10.1523/JNEUROSCI.4081-14.2015.
Development of Immunoassays for the Quantitative Assessment of Amyloid-β in the Presence of Therapeutic Antibody: Application to Pre-Clinical Studies.
IOS Press; DOI: 10.3233/JAD-142988.
Increasing the Receptor Tyrosine Kinase EphB2 Prevents Amyloid-β-induced Depletion of Cell-Surface Glutamate Receptors by a Mechanism that Requires EphB2′s PDZ-binding Motif and Neuronal Activity.
Journal of Biological Chemistry; doi: 10.1074/jbc.M115.666529.
NLRP3 Inflammasome Is Expressed and Functional in Mouse Brain Microglia but Not in Astrocytes.
PLOS ONE; DOI: 10.1371/journal.pone.0130624.
Development of Immunoassays for the Quantitative Assessment of Amyloid-β in the Presence of Therapeutic Antibody: Application to Pre-Clinical Studies.
ISO Press; DOI: 10.3233/JAD-142988.
A noninflammatory immune response in aged DNA Aβ42-immunized mice supports its safety for possible use as immunotherapy in AD patients..
Neurobiol. Aging; DOI: 10.1016/j.neurobiolaging.2014.12.011.
Analyzing and Modeling the Kinetics of Amyloid Beta Pores Associated with Alzheimer’s Disease Pathology.
PLOS One; DOI: 10.1371/journal.pone.0137357.
Photo-induced inhibition of Alzheimer's β-amyloid aggregation in vitro by rose bengal.
ScienceDirect; doi:10.1016/j.biomaterials.2014.10.058.
CREB expression mediates amyloid β-induced basal BDNF downregulation..
Neurobiol. Aging; doi: 10.1016/j.neurobiolaging.2015.04.014.