Kalirin Reduction Rescues Psychosis-Associated Behavioral Deficits in APPswe/PSEN1dE9 Transgenic Mice.
Neurobiology of Aging; http://dx.doi.org/10.1016/j.neurobiolaging.2017.02.006.
CD44 Splice Variants as Potential Players in Alzheimer’s Disease Pathology.
Journal of Alzheimer’s Disease; doi: 10.3233/JAD-161245.
In situ fibrillizing amyloid-beta 1-42 induces neurite degeneration and apoptosis of differentiated SH-SY5Y cells.
PLOS ONE; doi: 10.1371/journal.pone.0186636.
Analysis of the Impact of CD200 on Phagocytosis.
Mol Neurobiol.; doi: 10.1007/s12035-016-0223-6.
Role of microglial amylin receptors in mediating beta amyloid (Aβ)-induced inflammation.
Journal of Neuroinflammation; https://doi.org/10.1186/s12974-017-0972-9.
Application of quinazoline and pyrido[3,2-d]pyrimidine templates to design multi-targeting agents in Alzheimer's disease.
RSC Advances; DOI: 10.1039/C7RA02889J.
A human scFv antibody that targets and neutralizes high molecular weight pathogenic amyloid‐β oligomers.
J Neurochem; doi: 10.1111/jnc.14118.
In vitro study on potential pharmacological activity of curcumin analogues and their copper complexes.
Chem Biol Drug Des.; doi: 10.1111/cbdd.12847.
Amelioration of amyloid-β-induced deficits by DcR3 in an Alzheimer’s disease model.
BioMed Central; 10.1186/s13024-017-0173-0.
Development and Characterisation of a Novel NF-κB Reporter Cell Line for Investigation of Neuroinflammation.
Hindawi; https://doi.org/10.1155/2017/6209865.